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再灌注

网络reperfusion; Ischemia Reperfusion; Liver Ischemia Reperfusion

再灌注

再灌注

再灌注

reperfusion

...子通道激动剂在复氧（reoxygenation）或再灌注（reperfusion）之后会极大的减少体外缺氧缺糖缺血模型的神经元损伤，以 …

Ischemia Reperfusion

再者，短暂的高血压，能引出一个叫缺氧／再灌注（Ischemia Reperfusion）的问题，结果，一些细胞会因而死掉。若缺氧／再 …

Liver Ischemia Reperfusion

【摘要】：【目的】探讨在大鼠移植肝脏缺血/再灌注(Liver Ischemia Reperfusion)损伤动物模型,观察动物肝脏缺血/再灌注的组 …

IRI

...,缺血60 min+再灌注120 min;②缺血/再灌注(IRI)组:大鼠开胸后,继续观察40 min,然后予以缺血60 min+再灌注120 min;③假手 …

early reperfusion

...认识不断深化采用包括溶栓和介入性治疗在内的早期 再灌注（early reperfusion）治疗使AMI的死亡率进一步降低前言早在1…

GI-R

...鼠室旁核(PVN)注射Apelin-13对胃缺血.再灌注(GI-R)的损伤作用及其细胞机制.方法 PVN内微量注射Apelin-13,制备GI-R模型, …

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Conclusion : Ginkgo Biloba succi might have the protective effect on muscle cells during ischemia and reperfusion injury of limb.

结论：银杏叶提取液对缺血再灌注骨骼肌线粒体有保护作用。

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Conclusions : We conclude that postischemia mild hyperglycemia do not aggravate cerebral ischemia reperfusion injury .

结论：缺血后轻度高血糖对缺血再灌注损伤无明显影响。

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the effects of rapamycin as a pharmacological preconditioning agent during IR on the severity of injury in vitro have been studied .

想表达的中文意思：很少有研究探讨雷帕霉素作为一种预适应药物在在体缺血再灌注中的作用。

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Lidocaine attenuates cell injury induced by ischemic-reperfusion and inflammation , although the protective mechanisms are not understood.

利多卡因可减轻缺血再灌注和炎症反应所致的细胞损伤，其机制至今不明。

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CONCLUSION The results suggested that TL had a potent neuroprotective effect on cerebral ischemia and reperfusion in rats .

结论 TL 对大鼠缺血再灌注脂质过氧化脑损伤有一定的保护作用。

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Presently, few studies on the IRE1-mediated UPR signaling pathway in the model of cerebral ischemia and reperfusion had been reported.

目前在脑缺血再灌注模型中有关 IRE1 介导的 UPR 信号通路的研究较少见。

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Objective To investigate the effects of maltol on astrocyte proliferation in hippocampus after focal cerebral ischemia reperfusion in rats.

目的通过麦芽醇对脑缺血 -再灌注后星形胶质细胞增殖进行干预，以探讨其对胶质增生的影响。

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Bency: In the meantime , one method of pharmacological preconditioning was found to be helpful to reduce ischemia-reperfusion injury .

想表达的中文意思：同时发现，药物预适应可以显著地减少再灌注损伤！

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Electrocardiogram during the reperfusion phase and content of malondial-aldehyde in ischemia-reperfusion myocardium were analyzed .

分析再灌注期心电图变化及检测缺血心肌丙二醛含量。

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Objective To establish a experimental model for evaluating the remedy for denatured neurons after forebrain ischemia reperfusion .

目的：探索以变性神经元为观察对象，适用于脑缺血再灌注后干预实验的动物模型。

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